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AB10346

Sigma-Aldrich

Anti-SOD2 Antibody

serum, Chemicon®

Synonym(e):

Superoxide Dismutase 2 (SOD2)

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About This Item

UNSPSC-Code:
12352203
eCl@ss:
32160702
NACRES:
NA.41

Biologische Quelle

rabbit

Antikörperform

serum

Antikörper-Produkttyp

primary antibodies

Klon

polyclonal

Speziesreaktivität

mouse, human, rat

Hersteller/Markenname

Chemicon®

Methode(n)

western blot: suitable

NCBI-Hinterlegungsnummer

NM_000636

UniProt-Hinterlegungsnummer

P04179

Versandbedingung

wet ice

Posttranslationale Modifikation Target

unmodified

Angaben zum Gen

mouse ... Sod2(20656)

Allgemeine Beschreibung

This gene is a member of the iron/manganese superoxide dismutase family. It encodes a mitochondrial protein that forms a homotetramer and binds one manganese ion per subunit. This protein binds to the superoxide byproducts of oxidative phosphorylation and converts them to hydrogen peroxide and diatomic oxygen. Mutations in this gene have been associated with idiopathic cardiomyopathy (IDC), premature aging, sporadic motor neuron disease, and cancer. Alternate transcriptional splice variants, encoding different isoforms, have been characterized.

Immunogen

Epitope: AA G93 – Y217
Recombinant human SOD2

Anwendung

This Anti-SOD2 Antibody is validated for use in WB for the detection of SOD2.

Qualität

Routinely evaluated by immunoblot.

Zielbeschreibung

28 kDa

Rechtliche Hinweise

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

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Lagerklassenschlüssel

10 - Combustible liquids

WGK

WGK 1

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Loss of LRPPRC causes ATP synthase deficiency.
Mourier, A; Ruzzenente, B; Brandt, T; Kuhlbrandt, W; Larsson, NG
Human Molecular Genetics null
Anthony R Cyr et al.
Redox biology, 1, 172-177 (2013-09-12)
Manganese superoxide dismutase, encoded by the Sod2 gene, is a ubiquitously expressed mitochondrial antioxidant enzyme that is essential for mammalian life. Mice born with constitutive genetic knockout of Sod2 do not survive the neonatal stage, which renders the longitudinal study
Yukio Shimasaki et al.
Circulation research, 113(7), 891-901 (2013-07-04)
Mitochondria, although required for cellular ATP production, are also known to have other important functions that may include modulating cellular responses to environmental stimuli. However, the mechanisms whereby mitochondria impact cellular phenotype are not yet clear. To determine how mitochondria
Xiaoyuan Lin et al.
Aging and disease, 13(5), 1471-1487 (2022-10-04)
Excessive sodium fluoride (NaF) intake interferes with reproductive function in humans and animals; however, strategies to prevent these effects are still underexplored. Here, we showed that in vivo and in vitro supplementation of folic acid (FA) efficaciously improved the quality
S Baldelli et al.
Cell death & disease, 5, e1515-e1515 (2014-11-07)
Mitochondrial biogenesis and mitophagy are recognized as critical processes underlying mitochondrial homeostasis. However, the molecular pathway(s) coordinating the balance between these cellular programs is still poorly investigated. Here, we show an induction of the nuclear and mitochondrial peroxisome proliferator-activated receptor

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