S5394
Lipoprotein Deficient Serum from fetal calf
frozen liquid
Synonyme(s) :
Fetal Calf Serum, Lipoprotein Deficient Serum
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About This Item
Code UNSPSC :
12352207
Nomenclature NACRES :
NA.61
Source biologique:
bovine fetus
Forme:
frozen liquid
Conditions d'expédition:
dry ice
Produits recommandés
Source biologique
bovine fetus
Niveau de qualité
Forme
frozen liquid
Concentration
≥14 mg protein/mL Biuret
Technique(s)
cell culture | mammalian: suitable
cell culture | stem cell: suitable
Impuretés
<1 ng/mg protein endotoxin
<5 % of normal lipoprotein
Conditions d'expédition
dry ice
Température de stockage
−20°C
Description générale
Studies show that the production of sterols is reduced in human fibroblasts cultured in the presence of lipoprotein deficient serum.
Application
Lipoprotein Deficient Serum from fetal calf has been used as a supplement in culture media to grow Chinese hamster ovary (CHO)-derived cells, thymic lobes and human peripheral blood mononuclear cells (PBMCs).
Notes préparatoires
Aseptically filtered, 0.2 μm
Dialyzed against Dulbecco′s phosphate buffered saline, pH 7.4-7.5
Code de la classe de stockage
10 - Combustible liquids
Classe de danger pour l'eau (WGK)
WGK 3
Point d'éclair (°F)
Not applicable
Point d'éclair (°C)
Not applicable
Équipement de protection individuelle
Eyeshields, Gloves
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Les clients ont également consulté
P M Giles et al.
Clinica chimica acta; international journal of clinical chemistry, 113(2), 183-191 (1981-06-18)
The biosynthesis and efflux of sterols from cells into the medium were investigated in skin fibroblasts from a control, a patient with obligate heterozygous familial hypercholesterolaemia and a patient with the homozygous condition. The behaviour of the cells was studied
Brian T Emmer et al.
PLoS genetics, 17(1), e1009285-e1009285 (2021-01-30)
Hypercholesterolemia is a causal and modifiable risk factor for atherosclerotic cardiovascular disease. A critical pathway regulating cholesterol homeostasis involves the receptor-mediated endocytosis of low-density lipoproteins into hepatocytes, mediated by the LDL receptor. We applied genome-scale CRISPR screening to query the
Lipid-mediated presentation of MHC class II molecules guides thymocytes to the CD4 lineage
Komaniwa S, et al.
European Journal of Immunology, 39(1), 96-112 (2009)
Utsab Subedi et al.
International journal of molecular sciences, 24(3) (2023-02-12)
Neointimal hyperplasia is characterized by a loss of the contractile phenotype of vascular smooth muscle cells (VSMCs). Our group has recently shown that VSMC proliferation and migration are mediated by lysophosphatidic acid (LPA) during restenosis, but the role of autotaxin
Sterol regulatory element-binding protein (SREBP) cleavage regulates Golgi-to-endoplasmic reticulum recycling of SREBP cleavage-activating protein (SCAP)
Shao W and Espenshade PJ
The Journal of Biological Chemistry, 289(11), 7547-7557 (2014)
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