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Principaux documents

05-479

Sigma-Aldrich

Anti-Cytochrome C Antibody, clone C-7

ascites fluid, clone C-7, Upstate®

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About This Item

Code UNSPSC :
12352203
eCl@ss :
32160702

Source biologique

mouse

Forme d'anticorps

ascites fluid

Type de produit anticorps

primary antibodies

Clone

C-7, monoclonal

Espèces réactives

human, horse

Fabricant/nom de marque

Upstate®

Technique(s)

immunohistochemistry: suitable
western blot: suitable

Isotype

IgG

Numéro d'accès NCBI

Numéro d'accès UniProt

Conditions d'expédition

dry ice

Modification post-traductionnelle de la cible

unmodified

Informations sur le gène

human ... CYCS(54205)

Spécificité

Cytochrome c

Immunogène

Horse cytochrome c conjugated to KLH

Application

Detect Cytochrome C using this Anti-Cytochrome C Antibody, clone C-7 validated for use in WB, IH.
Research Category
Apoptosis & Cancer

Metabolism
Research Sub Category
Apoptosis - Additional

Enzymes & Biochemistry

Qualité

routinely evaluated by immunoblot on RIPA lysates from A431 cells

Description de la cible

14kDa

Liaison

Replaces: 04-1043

Forme physique

Ascites
mouse ascites containing 0.05% sodium azide

Stockage et stabilité

2 years at -20°C

Informations légales

UPSTATE is a registered trademark of Merck KGaA, Darmstadt, Germany

Clause de non-responsabilité

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Code de la classe de stockage

12 - Non Combustible Liquids

Classe de danger pour l'eau (WGK)

WGK 1

Point d'éclair (°F)

Not applicable

Point d'éclair (°C)

Not applicable


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Consulter la Bibliothèque de documents

Structural basis for the binding of an anti-cytochrome c antibody to its antigen: crystal structures of FabE8-cytochrome c complex to 1.8 A resolution and FabE8 to 2.26 A resolution.
Mylvaganam, S E, et al.
Journal of Molecular Biology, 281, 301-322 (1998)
J E Springer et al.
Nature medicine, 5(8), 943-946 (1999-07-30)
Traumatic spinal cord injury often results in complete loss of voluntary motor and sensory function below the site of injury. The long-term neurological deficits after spinal cord trauma may be due in part to widespread apoptosis of neurons and oligodendroglia
Rui Liu et al.
Molecular cancer therapeutics, 14(9), 2090-2102 (2015-07-05)
Tumor adaptive resistance to therapeutic radiation remains a barrier for further improvement of local cancer control. SIRT3, a member of the sirtuin family of NAD(+)-dependent protein deacetylases in mitochondria, promotes metabolic homeostasis through regulation of mitochondrial protein deacetylation and plays
So-Yeon Kim et al.
Oncotarget, 8(39), 64964-64973 (2016-08-05)
Small molecules to selectively induce cell death of undifferentiated human pluripotent stem cells (hPSCs) have been developed with the aim of lowering the risk of teratoma formation during hPSC-based cell therapy. In this context, we have reported that Quercetin (QC)
Xiao-Qing Ding et al.
Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology, 41(2), 835-848 (2017-02-20)
The present study investigated whether the transient receptor potential melastatin 4 (TRPM4) channel plays a role in high salt diet (HSD)-induced endothelial injuries. Western blotting and immunofluorescence were used to examine TRPM4 expression in the mesenteric endothelium of Dahl salt-sensitive

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