T1317
Eppendorf® Thermomixer Compact
with 1.5 mL block, AC/DC input 115 V AC
Synonym(s):
dry bath, heat block
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About This Item
UNSPSC Code:
41102404
NACRES:
NB.76
AC/DC input
115 V (AC)
manufacturer/tradename
Eppendorf® 5384000020 (USA)
availability
not available in EU
W × D × H
20.6 cm × 30.4 cm × 17.0 cm
capacity
, holds 1 block
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General description
This new thermomixer combines heating and shaking of 24 × 1.5 mL samples at a time to ease user workload. Incubation and mixing may also be performed independently. Includes autoclavable and freezer-safe numbered rack with 24 numbered positions make processing and transporting samples quick and easy.
- Short-mix function for quick vortex applications
- Mixing speed - 300 to 1,300 rpm
- Mixing stroke - 3 mm
- Temperature Control Range: 4°C above ambient up to 99°C in 1°C increments
- Temperature Accuracy - ± 0.5°C at 20 to 45 °C
Legal Information
Eppendorf is a registered trademark of Eppendorf AG
Certificates of Analysis (COA)
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Ni Li et al.
Molecular pharmacology, 91(4), 264-276 (2017-01-15)
Activation of liver X receptor (LXR) is associated with cholesterol metabolism and anti-inflammatory processes, which makes it beneficial to antiatherosclerosis therapy. Nevertheless, existing agonists that target LXR, for example TO901317, are related to unwanted side effects. In the present study
Mai Thi Nhu Tran et al.
Nature communications, 8(1), 1700-1700 (2017-11-24)
The transcription factor MafB is expressed by monocytes and macrophages. Efferocytosis (apoptotic cell uptake) by macrophages is important for inhibiting the development of autoimmune diseases, and is greatly reduced in Mafb-deficient macrophages. Here, we show the expression of the first
Monica de Gaetano et al.
Journal of inflammation (London, England), 12, 15-15 (2015-02-28)
Atherosclerosis, the underlying cause of heart attack and strokes, is a progresive dyslipidemic and inflammatory disease where monocyte-derived macrophage cells play a pivotal role. Although most of the mechanisms that contribute to the progression of atherosclerosis have been identified, there
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