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EMU028701

Sigma-Aldrich

MISSION® esiRNA

targeting mouse Dram1

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About This Item

UNSPSC Code:
41105324
NACRES:
NA.51

description

Powered by Eupheria Biotech

product line

MISSION®

form

lyophilized powder

esiRNA cDNA target sequence

TGATTGCCTGTGCTTCACTCATTTCTATAACCAAGCTGGAATGGAATCCAAAAGAAAAGGATTATATATATCACGTGGTGAGCGCCATCTGTGAGTGGACCGTGGCTTTTGGTTTTATTTTCTATTTCCTAACATTCATCCAAGATTTCCAGAGTGTCACTCTAAGGATATCCACAGAAATCAATGACGACTTTTGAAAGATCGAGAATCCTGTCTCATTCAGGGAGTGTCGCAGACAGTTTCTGGAAGTGGACAGAGGACGGACGGGCTTGGATGTCACCCTGATGGGGACTTTATCTGTGGCACATCCGGGACTTGAATTTCATTAAGAGTTCCTAGTAGTTCAATTTACAAAGGTATGTTTCCCTGGAGGATGGATAGCACCAACGACACTGTAGCAATATTTTTATATTTTCTAAAACAATCTTTTATGAACAAATTCATATGCAAAGAAGACGAGGCAT

Ensembl | mouse accession no.

ENSMUSG00000020057

NCBI accession no.

NM_027878

shipped in

ambient

storage temp.

−20°C

Gene Information

mouse ... DRAM1(71712) , 1200002N14Rik(71712)

General description

MISSION® esiRNA are endoribonuclease prepared siRNA. They are a heterogeneous mixture of siRNA that all target the same mRNA sequence. These multiple silencing triggers lead to highly-specific and effective gene silencing.

For additional details as well as to view all available esiRNA options, please visit SigmaAldrich.com/esiRNA.

Legal Information

MISSION is a registered trademark of Merck KGaA, Darmstadt, Germany

Storage Class Code

10 - Combustible liquids

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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K Liu et al.
Cell death & disease, 5, e1323-e1323 (2014-07-18)
Apoptosis-stimulating protein of p53-2 (ASPP2) induces apoptosis by promoting the expression of pro-apoptotic genes via binding to p53 or p73; however, the exact mechanisms by which ASPP2 induces apoptotic death in hepatoma cells are still unclear. Here, we show that
Brandon J Metge et al.
Scientific reports, 5, 11995-11995 (2015-07-07)
We have previously reported that expression of NMI (N-myc and STAT interactor) is compromised in invasive breast cancers. We also demonstrated that loss of NMI expression promotes epithelial-mesenchymal-transition and results in enhanced invasive ability of breast cancer cells. Additionally we

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