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163023

Sigma-Aldrich

1,1-Dichloroethylene

contains 200 ppm MEHQ as inhibitor, 99%

Synonym(s):

Vinylidene chloride

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About This Item

Linear Formula:
CH2=CCl2
CAS Number:
Molecular Weight:
96.94
Beilstein:
1733365
EC Number:
MDL number:
UNSPSC Code:
12162002
PubChem Substance ID:
NACRES:
NA.23

vapor density

3.46 (vs air)

vapor pressure

9.68 psi ( 20 °C)

Assay

99%

form

liquid

autoignition temp.

968 °F

contains

200 ppm MEHQ as inhibitor

expl. lim.

15.5 %

bp

30-32 °C (lit.)

mp

−122 °C (lit.)

density

1.213 g/mL at 20 °C (lit.)

storage temp.

2-8°C

SMILES string

ClC(Cl)=C

InChI

1S/C2H2Cl2/c1-2(3)4/h1H2

InChI key

LGXVIGDEPROXKC-UHFFFAOYSA-N

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Application

1,1-Dichloroethylene can be used as a monomer to synthesize polymercomposite latexes by emulsion polymerization.

Signal Word

Danger

Hazard Classifications

Acute Tox. 3 Oral - Acute Tox. 4 Inhalation - Carc. 2 - Eye Irrit. 2 - Flam. Liq. 1 - Skin Irrit. 2

Storage Class Code

3 - Flammable liquids

WGK

WGK 3

Flash Point(F)

-2.2 °F

Flash Point(C)

-19 °C

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

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Fan Chen et al.
Frontiers in microbiology, 9, 2306-2306 (2018-10-17)
Bioelectrochemical systems (BESs) are regarded as a promising approach for the enhanced dechlorination of chlorinated aliphatic hydrocarbons (CAHs). However, the electron distribution and transfer considering dechlorination, methanogenesis, and other bioprocesses in these systems are little understood. This study investigated the
Xin Ma et al.
Water research, 40(6), 1155-1166 (2006-02-28)
Column experiments were performed to investigate a technology for remediating aquifers contaminated with chlorinated solvents. The technology involves installation of hollow-fiber membranes in the subsurface to supply hydrogen gas (H2) to groundwater to support biological reductive dechlorination in situ. Three
Juliet A Jones et al.
Chemical research in toxicology, 16(10), 1306-1317 (2003-10-21)
A proteome profiling approach was used to compare effects of two toxicants, 1,1-dicloroethylene (DCE) and diclofenac, which covalently adduct hepatic proteins. Bile was examined as a potential source of protein alterations since both toxicants target the hepatic biliary canaliculus. Bile
Erik J Martin et al.
The Journal of pharmacology and experimental therapeutics, 304(1), 121-129 (2002-12-20)
Hepatotoxicity induced by 1,1-dichloroethylene (DCE) is mediated by cytochrome P450-dependent metabolism to reactive intermediates, including the epoxide. We have tested the hypothesis that mitochondria are a primary target of toxicity by investigating dose- and time-dependent effects of DCE on mitochondrial
Masarin Ban et al.
Toxicology, 184(1), 41-50 (2002-12-31)
Using immunotoxic functional tests, namely IgM response to sheep red blood cells (SRBCs) and interferon-gamma (IFN-gamma) production, this study simultaneously evaluated the effects of inhaled chloroform (10, 20, and 50 ppm), carbon tetrachloride (100, 200, and 300 ppm), 1,1-dichloroethylene (5

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