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Key Documents

SAB2501406

Sigma-Aldrich

Anti-GABRG2 antibody produced in goat

affinity isolated antibody, buffered aqueous solution

Synonym(s):

Anti-CAE2, Anti-ECA2, Anti-GEFSP3, Gamma-aminobutyric acid (GABA) A receptor, gamma 2, Gamma-aminobutyric acid A receptor

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About This Item

UNSPSC Code:
12352203
NACRES:
NA.41

biological source

goat

Quality Level

conjugate

unconjugated

antibody form

affinity isolated antibody

antibody product type

primary antibodies

clone

polyclonal

form

buffered aqueous solution

species reactivity

bovine, mouse, rat, human

technique(s)

indirect ELISA: suitable
western blot: suitable

UniProt accession no.

shipped in

dry ice

storage temp.

−20°C

target post-translational modification

unmodified

Gene Information

human ... GABRG2(2566)

Immunogen

Peptide with sequence NNATHLQERDE, from the internal region of the protein sequence according to NP_944494.1; NP_000807.2; NP_944493.2

Features and Benefits

Evaluate our antibodies with complete peace of mind. If the antibody does not perform in your application, we will issue a full credit or replacement antibody. Learn more.

Physical form

Supplied at 0.5 mg/mL in 20mM Tris (pH 7.3) and 150mM NaCl with 0.02% sodium azide and 0.5% bovine serum albumin.

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Storage Class Code

10 - Combustible liquids

WGK

WGK 2

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable


Certificates of Analysis (COA)

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Zheng Wu et al.
Nature communications, 5, 4159-4159 (2014-06-14)
Amyloid plaques and tau tangles are common pathological hallmarks for Alzheimer's disease (AD); however, reducing Aβ production failed to relieve the symptoms of AD patients. Here we report a high GABA (γ-aminobutyric acid) content in reactive astrocytes in the dentate
Ming Gao et al.
The Journal of neuroscience : the official journal of the Society for Neuroscience, 34(32), 10770-10779 (2014-08-08)
The developmental increase in the strength of inhibitory synaptic circuits defines the time window of the critical period for plasticity in sensory cortices. Conceptually, plasticity of inhibitory synapses is an attractive mechanism to allow for homeostatic adaptation to the sensory

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