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Oncogenic PIK3CA induces centrosome amplification and tolerance to genome doubling.

Nature communications (2017-11-25)
Inma M Berenjeno, Roberto Piñeiro, Sandra D Castillo, Wayne Pearce, Nicholas McGranahan, Sally M Dewhurst, Valerie Meniel, Nicolai J Birkbak, Evelyn Lau, Laurent Sansregret, Daniele Morelli, Nnennaya Kanu, Shankar Srinivas, Mariona Graupera, Victoria E R Parker, Karen G Montgomery, Larissa S Moniz, Cheryl L Scudamore, Wayne A Phillips, Robert K Semple, Alan Clarke, Charles Swanton, Bart Vanhaesebroeck
ZUSAMMENFASSUNG

Mutations in PIK3CA are very frequent in cancer and lead to sustained PI3K pathway activation. The impact of acute expression of mutant PIK3CA during early stages of malignancy is unknown. Using a mouse model to activate the Pik3ca

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Marke
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Poly-L-Lysin -Lösung, 0.1 % (w/v) in H2O
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Ziegenserum
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Esel-Serum
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(Z)-4-Hydroxytamoxifen, ≥98% Z isomer
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Triton X-100, laboratory grade
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Albumin aus Rinderserum, heat shock fraction, pH 7, ≥98%
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Anti-phospho-Histon H2A.X (Ser139)-Antikörper, Klon JBW301, clone JBW301, Upstate®, from mouse
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Anti-β-Actin-Antikörper, Maus monoklonal, clone AC-15, purified from hybridoma cell culture
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Nocodazol, ≥99% (TLC), powder
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Histochemisches Färbungskit für Seneszenzzellen, sufficient for 100 tests
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Anti-α-Tubulin-Antikörper, monoklonaler Antikörper der Maus, clone B-5-1-2, purified from hybridoma cell culture
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Ribonuklease A aus Rinderpankreas, Type I-AS, 50-100 Kunitz units/mg protein
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Trypsin -Lösung aus Schweinepankreas, 1 ×, sterile-filtered, BioReagent, suitable for cell culture, 2.5 g porcine trypsin per liter in Hanks′ Balanced Salt Solution with phenol red
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Monoclonal Anti-α-Actinin antibody produced in mouse, clone BM-75.2, ascites fluid
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Dihydrocytochalasin B