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Merck

205435

Sigma-Aldrich

May-Grünwald Stain

Suitable for the differential staining of cellular elements of blood

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About This Item

MDL number:
UNSPSC Code:
12171500
NACRES:
NA.47

Quality Level

form

powder

color

green to black

λmax

522 nm (2nd)
645 nm

application(s)

diagnostic assay manufacturing
hematology
histology

storage temp.

room temp

SMILES string

COC1=NC(=NC2=C1N=CN2C3C(C(C(O3)CO)O)O)N

InChI

InChI=1S/C11H15N5O5/c1-20-9-5-8(14-11(12)15-9)16(3-13-5)10-7(19)6(18)4(2-17)21-10/h3-4,6-7,10,17-19H,2H2,1H3,(H2,12,14,15)/t4-,6+,7?,10-/m1/s1

InChI key

IXOXBSCIXZEQEQ-PKJMTWSGSA-N

Categorías relacionadas

General description

May-Grünwald stain is a Romanowsky neutraldye stain based on the combination ofmethylene blue azures as the basic dyecomponent and eosin as the acid dyecomponent.It is intended for use in staining blood films orbone marrow films. It is used routinely for diagnostic purposes in hematology.

Application

May-Grünwald′s stain is used for the differential staining of cellular elements of blood.

pictograms

Exclamation mark

signalword

Warning

Hazard Classifications

Acute Tox. 4 Oral - Eye Irrit. 2 - Skin Sens. 1

Storage Class

11 - Combustible Solids

wgk_germany

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

ppe

dust mask type N95 (US), Eyeshields, Gloves


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Saito, Hidehiko and Kunishima, Shinji
American Journal of Hematology, 83(4) (2008)
Epidemiological characteristics of Malassezia folliculitis and use of the May-Grunwald-Giemsa stain to diagnose the infection
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Fucoidan is a unique bioactive and dietary polymer enriched mainly in the cell wall matrix of the brown seaweeds. This present study was intended to reveal the antigenotoxicity effect of fucoidan on 4-nitroquinolin-1-oxide (4-NQO) induced genetics damage and apoptosis in
Michael L Megison et al.
PloS one, 9(1), e86843-e86843 (2014-02-06)
Recently, investigators showed that mice with syngeneic murine gliomas that were treated with a neuroattenuated oncolytic herpes simplex virus-1 (oHSV), M002, had a significant increase in survival. M002 has deletions in both copies of the γ134.5 gene, enabling replication in

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