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Dextran from Leuconostoc spp.

Mr 450,000-650,000

Linear Formula:
CAS Number:
EC Number:
MDL number:

Quality Level

mol wt

Mr 450,000-650,000


≤7% loss on drying


≤0.05 at 375 nm in H2O at 10%

storage temp.

room temp

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Dextran is a branched glucan composed of linear α(1→6) linked glucose units and α (1→3) link initiated branches. Dextran ranges in size from 10,000 to 150,000 Kd. Dextrans are used in many applications as volume extenders, stabilizers, matrix components, binding platforms, lubricants and physical structure components. Dextran-500 is used to simulate erythrocyte aggregation in cardiovascular research.

Storage Class Code

11 - Combustible Solids



Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

Certificate of Analysis

Certificate of Origin

Peng Kai Ong et al.
American journal of physiology. Heart and circulatory physiology, 298(6), H1870-H1878 (2010-03-30)
Formation of a cell-free layer is an important dynamic feature of microcirculatory blood flow, which can be influenced by rheological parameters, such as red blood cell aggregation and flow rate. In this study, we investigate the effect of these two
Martí Ortega-Ribera et al.
Biotechnology and bioengineering, 115(10), 2585-2594 (2018-06-26)
Maintenance of the complex phenotype of primary hepatocytes in vitro represents a limitation for developing liver support systems and reliable tools for biomedical research and drug screening. We herein aimed at developing a biosystem able to preserve human and rodent
Marie-Elena Brett et al.
Integrative biology : quantitative biosciences from nano to macro, 10(4), 242-252 (2018-04-07)
Numerous studies have demonstrated the importance of altered hyaluronan metabolism to malignant progression of multiple tumor types, including breast carcinomas. Increased hyaluronan (HA) metabolism in the stroma of primary tumors promotes activation of oncogenic signaling pathways that impact tumor initiation
Young-Chan Kim et al.
Nature chemical biology, 15(9), 907-916 (2019-08-21)
Toll-like receptor (TLR)/myeloid differentiation primary response protein (MYD88) signaling aggravates sepsis by impairing neutrophil migration to infection sites. However, the role of intracellular fatty acids in TLR/MYD88 signaling is unclear. Here, inhibition of fatty acid synthase by C75 improved neutrophil
Yang Yang et al.
Biochimica et biophysica acta, 1840(1), 288-293 (2013-10-01)
Abnormal adhesion of red blood cells (RBCs) to vascular endothelium is often associated with reduced levels of sialic acids on RBC membranes and with elevated levels of pro-adhesive plasma proteins. However, the synergistic effects of these two factors on the

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