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Merck
  • MicroRNA-128-3p regulates mitomycin C-induced DNA damage response in lung cancer cells through repressing

MicroRNA-128-3p regulates mitomycin C-induced DNA damage response in lung cancer cells through repressing

Oncotarget (2017-09-25)
Rui Zhang, Chang Liu, Yahan Niu, Ying Jing, Haiyang Zhang, Jin Wang, Jie Yang, Ke Zen, Junfeng Zhang, Chen-Yu Zhang, Donghai Li
摘要

The DNA damage response is critical for maintaining genome integrity and preventing damage to DNA due to endogenous and exogenous insults. Mitomycin C (MMC), a potent DNA cross-linker, is used as a chemotherapeutic agent because it causes DNA inter-strand cross-links (DNA ICLs) in cancer cells. While many microRNAs, which may serve as oncogenes or tumor suppressors, are grossly dysregulated in human cancers, little is known about their roles in MMC-treated lung cancer. Here, we report that miR-128-3p can attenuate repair of DNA ICLs by targeting

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甲基镁碳酸酯 溶液, 2.0 M in DMF
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MISSION® esiRNA, targeting human SPTAN1