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Merck
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文件

SAB1305552

Sigma-Aldrich

单克隆抗-LC3（APG8） 小鼠抗

clone 166AT1234, IgG fraction of antiserum, buffered aqueous solution

别名:

MAP1 轻链 3 样蛋白 1, MAP1LC3A, 微管相关蛋白 1A/1B 轻链 3A, 自噬相关泛素样修饰物 LC3 A, 自噬相关蛋白 LC3 A

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About This Item

分類程式碼代碼:
12352203
NACRES:
NA.41

生物源

mouse

品質等級

100

抗體表格

IgG fraction of antiserum

抗體產品種類

primary antibodies

無性繁殖

166AT1234, monoclonal

形狀

buffered aqueous solution

分子量

14272 Da

物種活性

rat, mouse, human

技術

immunofluorescence: 1:25
immunohistochemistry: 1:50-1:100
western blot: 1:1000

同型

IgG1κ

NCBI登錄號

NP_115903.1

UniProt登錄號

Q9H492

運輸包裝

wet ice

儲存溫度

−20°C

目標翻譯後修改

unmodified

基因資訊

human ... MAP1LC3A(84557)

外觀

以含有 0.09％（W/V）叠氮化钠的 PBS 形式提供

免責聲明

除非我们的产品目录或产品附带的其他公司文档另有说明，否则我们的产品仅供研究使用，不得用于任何其他目的，包括但不限于未经授权的商业用途、体外诊断用途、离体或体内治疗用途或任何类型的消费或应用于人类或动物。

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儲存類別代碼

10 - Combustible liquids

閃點（°F）

Not applicable

閃點（°C）

Not applicable

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Xiaoming Shu et al.
Molecular medicine reports, 16(2), 1180-1188 (2017-06-07)
Peripheral blood T lymphocytopenia has previously been identified in polymyositis/dermatomyositis (PM/DM) patients. Therefore, the present study aimed to examine the potential role of autophagy in peripheral blood T cell survival in PM/DM patients. Transmission electron microscopy was used to detect
Maomao Sun et al.
Frontiers in immunology, 12, 685523-685523 (2021-08-03)
Recent studies have shown that autophagy upregulation can attenuate sepsis-induced acute kidney injury (SAKI). The tumor suppressor p53 has emerged as an autophagy regulator in various forms of acute kidney injury (AKI). Our previous studies showed that p53 acetylation exacerbated
Rongrong Hua et al.
International journal of molecular sciences, 19(5) (2018-05-08)
We have reported that conventional protein kinase Cγ (cPKCγ)-modulated neuron-specific autophagy improved the neurological outcome of mice following ischemic stroke through the Akt-mechanistic target of rapamycin (mTOR) pathway. However, its detailed molecular mechanism remains unclear. In this study, primary cortical
Rongrong Hua et al.
Journal of cellular biochemistry, 120(9), 15915-15923 (2019-05-14)
The sequential reactivation of mechanistic target of rapamycin (mTOR) inhibited autophagic flux in neurons exposed to oxygen-glucose deprivation/reperfusion (OGD/R), which was characterized by reduction of autophagosome formation and restriction of autolysosome degradation. However, its detailed molecular mechanism was still unknown.

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