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Combined replacement effects of human modified β-hexosaminidase B and GM2 activator protein on GM2 gangliosidoses fibroblasts.

Biochemistry and biophysics reports (2016-06-08)
Keisuke Kitakaze, Chikako Tasaki, Youichi Tajima, Takatsugu Hirokawa, Daisuke Tsuji, Hitoshi Sakuraba, Kohji Itoh
RESUMEN

GM2 gangliosidoses are autosomal recessive lysosomal storage diseases (LSDs) caused by mutations in the

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Anti-β-actina monoclonal antibody produced in mouse, clone AC-74, ascites fluid